The effects of exercise on myocardial capillary bed and connective tissue in senescent rats
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The effects of exercise on myocardial capillary bed and connective tissue in senescent rats

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Published .
Written in English

Subjects:

  • Heart -- Aging,
  • Exercise -- Physiological aspects,
  • Rats -- Physiology

Book details:

Edition Notes

Statementby Merry B. Finch
The Physical Object
FormatMicroform
Paginationvii, 76 leaves
Number of Pages76
ID Numbers
Open LibraryOL14056502M

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Using papillary muscle preparations, aging has been associated to functional, biochemical, and electric performance alterations of the heart. 1 Cardiac senescence is characterized by functional lowering of myocardial strength and contraction speed, 26,27 prolongation of the relaxation phase and stiffening of both myocytes and mural connective tissue. 28–30 Although these changes associated with age are expected to affect LV systolic function Cited by: Effect of exercise on myocardial infarction in young vs. old male rats: electrocardiograph changes Bernard C. Wexler, Ph.D.* Bruce P. Greenberg, Ph.D.** Cincinnati, Ohio Death due to myocardial infarction is reaching epidemic proportions and its age of onset is no longer confined to the elderly but is now affecting young adults in ever increasing grupo-operativo-gei-porcino.com by: 9. Effect of Exercise on Acute Myocardial Infarction in Rats JUDITH S. HOCHMAN, MD, FACC, BERNADINE HEALY, MD, FACC Baltimore, Maryland Infarct expansion, the time-related thinning and dilation of an acute transmural infarct, leads to aneurysm for• mation and cardiac rupture in . The effects of exercise on myocardial capillary bed and connective tissue in senescent rats by Merry B. Finch.

Dec 01,  · The effects of a cholesterol-rich diet and exercise training on the myocardial capillary network and capillary ultrastructure were examined using Sprague-Dawley rats subjected to a 7-week exhaustive swimming scheme. A total of 16 animals were randomly divided into four groups consisting of normal dietinactive, normal dietexercise, cholesterol dietinactive, and cholesterol dietexercise Cited by: ie. connective tissue, vascular and interstitial space, significantly (P senescent rats. The knowledge gained from the current investigation will further the information already available on the effects of ageing on the myocardium. The results will be discussed in terms of human myocardial ageing. iv. What is the ability of myocardial tissue to PROPAGATE electrical impulses? During exercise, cardiac output increases dramatically, but mean arterial blood pressure rises very little. WHY? What is the role of the dense connective tissue termed anuli bibrosis cordis in the function of the heart? Specifically, at equivalent levels of exercise-induced muscle hyperemia, V RBC and F RBC within individual capillaries must be elevated in senescent vs. younger rats. Consequently, for each capillary, the ratio Do 2 /βQ̇ or, more correctly, Do 2 /β V RBC, will fall to a greater extent in senescent grupo-operativo-gei-porcino.com by:

The effects of aging on capillary hemodynamics in contracting rat spinotrapezius muscle Article in Microvascular Research 77(2) · December with 57 Reads How we measure 'reads'. Changes of Myocardial Structure with Aging. Authors; Authors and affiliations Effects of age and exercise on the extent of the myocardial capillary bed. Anat Ree 55–62 CrossRef Google Scholar. Tomanek RJ, Karlsson UL () Myocardial ultrastructure in young and senescent rats. J Ultrastruct Res – PubMed CrossRef Google Author: H. Frenzel, R. Zweihoff, B. Schwartzkopff, K.-F. Bürrig. Mathematically modelling the electrical activity of the heart: from cell to body surface and back again / Andrew J. Pullan, Martin L. Buist, Leo K. Cheng. Cardiac Implications after Myocardial Infarction in Rats previously Undergoing Physical Exercise Eduardo Carvalho de Arruda Veiga1, Leslie Andrews Portes1,2, Danilo Sales Bocalini1,5, Ednei Luiz Antonio3, Alexandra Alberta dos Santos 1, Marília Higuchi Santos4, Flavio André Silva, Paulo José Ferreira Tucci1,3 Departamento de Medicina – Disciplina de Cardiologia – UNIFESP1, São Paulo.